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DESCRIBE <http://purl.uniprot.org/SHA-384/012F3359D0816C3B387AFE65BD005E9F6C8F851F08F4729AE16F0C8D72A5B4B393F7FC7502691A6ACD3A83C057D0F70F>
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http://purl.uniprot.org/SHA-384/012F3359D0816C3B387AFE65BD005E9F6C8F851F08F4729AE16F0C8D72A5B4B393F7FC7502691A6ACD3A83C057D0F70F
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/012F3359D0816C3B387AFE65BD005E9F6C8F851F08F4729AE16F0C8D72A5B4B393F7FC7502691A6ACD3A83C057D0F70F
http://www.w3.org/2000/01/rdf-schema#comment
"Data from knockout mice suggest that Nod2 inhibits cardiac hypertrophy; Nod2 appears to be involved in dominant signaling pathways leading to (or preventing) cardiac hypertrophy and fibrosis in response to hypertrophic stimuli."
xsd:string
http://purl.uniprot.org/uniprot/#_5488DCE96E4911F2CFB791D2C57722E03041DBF715A78C4E8FACDF8E7634F4C5A06E0C48995DCAC12D20036ED909A14C
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/012F3359D0816C3B387AFE65BD005E9F6C8F851F08F4729AE16F0C8D72A5B4B393F7FC7502691A6ACD3A83C057D0F70F
http://purl.uniprot.org/uniprot/Q8K3Z0
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/012F3359D0816C3B387AFE65BD005E9F6C8F851F08F4729AE16F0C8D72A5B4B393F7FC7502691A6ACD3A83C057D0F70F
http://purl.uniprot.org/uniprot/#_Q8K3Z0-mappedCitation-23958879
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/012F3359D0816C3B387AFE65BD005E9F6C8F851F08F4729AE16F0C8D72A5B4B393F7FC7502691A6ACD3A83C057D0F70F