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DESCRIBE <http://purl.uniprot.org/SHA-384/019B30B39A81008C26387FFF92AF6E191B65DD6FC4FB1514497FC2EED4F1E9D4AC8C7886F9B6911B935DB1169D9E8D86>
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http://purl.uniprot.org/SHA-384/019B30B39A81008C26387FFF92AF6E191B65DD6FC4FB1514497FC2EED4F1E9D4AC8C7886F9B6911B935DB1169D9E8D86
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/019B30B39A81008C26387FFF92AF6E191B65DD6FC4FB1514497FC2EED4F1E9D4AC8C7886F9B6911B935DB1169D9E8D86
http://www.w3.org/2000/01/rdf-schema#comment
"Data suggest that GEP100-Arf6-AMAP1-cortactin pathway activated by VEGFR2 appears to be common in angiogenesis and cancer invasion and metastasis and provides their new therapeutic targets."
xsd:string
http://purl.uniprot.org/uniprot/#_DD79553ADD655FB93FD4752314AF7EC93EB0342C87E94A83FFC01A4892316104A83FB29E344AE65ABC65AE5846ECD852
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/019B30B39A81008C26387FFF92AF6E191B65DD6FC4FB1514497FC2EED4F1E9D4AC8C7886F9B6911B935DB1169D9E8D86
http://purl.uniprot.org/uniprot/A0A0A0MRE5
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/019B30B39A81008C26387FFF92AF6E191B65DD6FC4FB1514497FC2EED4F1E9D4AC8C7886F9B6911B935DB1169D9E8D86
http://purl.uniprot.org/uniprot/#_A0A0A0MRE5-mappedCitation-21858086
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/019B30B39A81008C26387FFF92AF6E191B65DD6FC4FB1514497FC2EED4F1E9D4AC8C7886F9B6911B935DB1169D9E8D86