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DESCRIBE <http://purl.uniprot.org/SHA-384/01A15D63C6CA053DAF26598FFD586C6B053EAAF22923A9C4EC18A2D8E76018A27B5D6DFD70FF6D39B50711CE2137A172>
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http://purl.uniprot.org/SHA-384/01A15D63C6CA053DAF26598FFD586C6B053EAAF22923A9C4EC18A2D8E76018A27B5D6DFD70FF6D39B50711CE2137A172
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/01A15D63C6CA053DAF26598FFD586C6B053EAAF22923A9C4EC18A2D8E76018A27B5D6DFD70FF6D39B50711CE2137A172
http://www.w3.org/2000/01/rdf-schema#comment
"Our studies demonstrate a primary LQT-3 (Na+ channel) defect not influenced by the KCNH2 polymorphism and drug interactions unique to the proband that correlate with improved management of arrhythmias in the patient."
xsd:string
http://purl.uniprot.org/uniprot/#_0AF5D995C118B7263031468E420A6A66D49A30E14E0B451F345BA230754FC3FC6C2E0F7E786357F7E38184D383E34060
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/01A15D63C6CA053DAF26598FFD586C6B053EAAF22923A9C4EC18A2D8E76018A27B5D6DFD70FF6D39B50711CE2137A172
http://purl.uniprot.org/uniprot/Q86V90
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/01A15D63C6CA053DAF26598FFD586C6B053EAAF22923A9C4EC18A2D8E76018A27B5D6DFD70FF6D39B50711CE2137A172
http://purl.uniprot.org/uniprot/#_Q86V90-mappedCitation-23277474
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/01A15D63C6CA053DAF26598FFD586C6B053EAAF22923A9C4EC18A2D8E76018A27B5D6DFD70FF6D39B50711CE2137A172