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DESCRIBE <http://purl.uniprot.org/SHA-384/0426C92FC1B5F706D03B4D6341C26275011638B1D441FEFA8C66FC7AE78A642CFD03A56A42229988F15932039994D96D>
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http://purl.uniprot.org/SHA-384/0426C92FC1B5F706D03B4D6341C26275011638B1D441FEFA8C66FC7AE78A642CFD03A56A42229988F15932039994D96D
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/0426C92FC1B5F706D03B4D6341C26275011638B1D441FEFA8C66FC7AE78A642CFD03A56A42229988F15932039994D96D
http://www.w3.org/2000/01/rdf-schema#comment
"results unravel an instrumental role of ATP13A2 deficiency on lysosomal function and cell viability and demonstrate the feasibility and therapeutic potential of modulating ATP13A2 levels in the context of PD"
xsd:string
http://purl.uniprot.org/uniprot/#_BC538D23B46C01CBE8388DFB3B3B9B130357246F53E19E0DCBCE47FCFA5E2093B11CD5AD2ED8AFBB8C5F460D5854AE42
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/0426C92FC1B5F706D03B4D6341C26275011638B1D441FEFA8C66FC7AE78A642CFD03A56A42229988F15932039994D96D
http://purl.uniprot.org/uniprot/Q52PK6
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/0426C92FC1B5F706D03B4D6341C26275011638B1D441FEFA8C66FC7AE78A642CFD03A56A42229988F15932039994D96D
http://purl.uniprot.org/uniprot/#_Q52PK6-mappedCitation-22647602
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/0426C92FC1B5F706D03B4D6341C26275011638B1D441FEFA8C66FC7AE78A642CFD03A56A42229988F15932039994D96D