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http://purl.uniprot.org/SHA-384/04F0E7F7AE092E2E7544D4FDD813E60EE710D754D31BAF2B3977EC5CF7AFBE42521A52DA0DE849ABF5B2D678042B73CDhttp://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/04F0E7F7AE092E2E7544D4FDD813E60EE710D754D31BAF2B3977EC5CF7AFBE42521A52DA0DE849ABF5B2D678042B73CDhttp://www.w3.org/2000/01/rdf-schema#comment"A -588T polymorphism of the GCLM gene causes a decrease in endothelial NO bioactivity causing impairment of endothelium-dependent vasomotor function in large and resistance coronary arteries. The GCL-GSH-NO axis may defend against coronary artery disease; Observational study of gene-disease association. (HuGE Navigator)"xsd:string
http://purl.uniprot.org/uniprot/#_45527DB114A53563E07B9326F6FEC6EBA9490BF9474B9EE0FF53C26FA3C491EAFC8F5345FB63897BE9039A501AA0338Ehttp://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/04F0E7F7AE092E2E7544D4FDD813E60EE710D754D31BAF2B3977EC5CF7AFBE42521A52DA0DE849ABF5B2D678042B73CD
http://purl.uniprot.org/uniprot/Q99711http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/04F0E7F7AE092E2E7544D4FDD813E60EE710D754D31BAF2B3977EC5CF7AFBE42521A52DA0DE849ABF5B2D678042B73CD
http://purl.uniprot.org/uniprot/#_Q99711-mappedCitation-12975258http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/04F0E7F7AE092E2E7544D4FDD813E60EE710D754D31BAF2B3977EC5CF7AFBE42521A52DA0DE849ABF5B2D678042B73CD