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DESCRIBE <http://purl.uniprot.org/SHA-384/05AF4E643159CB19ABC34206AC31DDC289107702A99F4CE766D11230DF0F427F59069E04E8C7F2882D28DD4AEDCFB4A9>
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http://purl.uniprot.org/SHA-384/05AF4E643159CB19ABC34206AC31DDC289107702A99F4CE766D11230DF0F427F59069E04E8C7F2882D28DD4AEDCFB4A9
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/05AF4E643159CB19ABC34206AC31DDC289107702A99F4CE766D11230DF0F427F59069E04E8C7F2882D28DD4AEDCFB4A9
http://www.w3.org/2000/01/rdf-schema#comment
"SOCS3 critically controls the phenotype and function of macrophages and neutrophils under inflammatory conditions and loss of SOCS3 promotes the angiogenic phenotype of the cells through up-regulation of arginase-1."
xsd:string
http://purl.uniprot.org/uniprot/#_46E5942AD1E31B1AF7841FA08A984D001ACCDDC8EE22A8EE24BC5DBE0AA92C3D001DC72B326EADDA18A32C2059D91090
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/05AF4E643159CB19ABC34206AC31DDC289107702A99F4CE766D11230DF0F427F59069E04E8C7F2882D28DD4AEDCFB4A9
http://purl.uniprot.org/uniprot/O35718
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/05AF4E643159CB19ABC34206AC31DDC289107702A99F4CE766D11230DF0F427F59069E04E8C7F2882D28DD4AEDCFB4A9
http://purl.uniprot.org/uniprot/#_O35718-mappedCitation-29452101
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/05AF4E643159CB19ABC34206AC31DDC289107702A99F4CE766D11230DF0F427F59069E04E8C7F2882D28DD4AEDCFB4A9