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DESCRIBE <http://purl.uniprot.org/SHA-384/08BF142FF8315056371E7BA4E400E57F3AD8CAC27E547C019D10ED5DFAAFA7CAB35222A4AE7C582A336B86C875248D51>
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http://purl.uniprot.org/SHA-384/08BF142FF8315056371E7BA4E400E57F3AD8CAC27E547C019D10ED5DFAAFA7CAB35222A4AE7C582A336B86C875248D51
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/08BF142FF8315056371E7BA4E400E57F3AD8CAC27E547C019D10ED5DFAAFA7CAB35222A4AE7C582A336B86C875248D51
http://www.w3.org/2000/01/rdf-schema#comment
"Data show that in angiotensin II type 1a receptor knockout mice mRNA expression and immunostaining of ACE2 and Mas in the injured artery were greater with less neointimal formation than in wild-type mice."
xsd:string
http://purl.uniprot.org/uniprot/#_0CE1A44AE703BB3E28EA94D26136CD63A35C947CFF152D4B5365AEEF0015C004BD1473F5F83F394B6526E43D798EA088
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/08BF142FF8315056371E7BA4E400E57F3AD8CAC27E547C019D10ED5DFAAFA7CAB35222A4AE7C582A336B86C875248D51
http://purl.uniprot.org/uniprot/Q8R0I0
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/08BF142FF8315056371E7BA4E400E57F3AD8CAC27E547C019D10ED5DFAAFA7CAB35222A4AE7C582A336B86C875248D51
http://purl.uniprot.org/uniprot/#_Q8R0I0-mappedCitation-22665126
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/08BF142FF8315056371E7BA4E400E57F3AD8CAC27E547C019D10ED5DFAAFA7CAB35222A4AE7C582A336B86C875248D51