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DESCRIBE <http://purl.uniprot.org/SHA-384/0A6C9B853F3DF864ADADDA6F6F0FE83C6732A2146D88ED1D046099C8D02DBB03CFCE846E32A12A6B8DDF498F8B2F49A0>
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http://purl.uniprot.org/SHA-384/0A6C9B853F3DF864ADADDA6F6F0FE83C6732A2146D88ED1D046099C8D02DBB03CFCE846E32A12A6B8DDF498F8B2F49A0
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/0A6C9B853F3DF864ADADDA6F6F0FE83C6732A2146D88ED1D046099C8D02DBB03CFCE846E32A12A6B8DDF498F8B2F49A0
http://www.w3.org/2000/01/rdf-schema#comment
"decreased levels of AMBP-1 play a critical role in producing vascular AM (adrenomedullin) hyporesponsiveness during the late stage of sepsis (AMBP-1)"
xsd:string
http://purl.uniprot.org/uniprot/#_4E4383EC0F2CBACB257A61E569DEB9C07CBA3C7D84C94EDC88B1E068C492DB08F99D04DFB26139E7E0600F5F1DC07ABB
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/0A6C9B853F3DF864ADADDA6F6F0FE83C6732A2146D88ED1D046099C8D02DBB03CFCE846E32A12A6B8DDF498F8B2F49A0
http://purl.uniprot.org/uniprot/P35318
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/0A6C9B853F3DF864ADADDA6F6F0FE83C6732A2146D88ED1D046099C8D02DBB03CFCE846E32A12A6B8DDF498F8B2F49A0
http://purl.uniprot.org/uniprot/#_P35318-mappedCitation-12643861
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/0A6C9B853F3DF864ADADDA6F6F0FE83C6732A2146D88ED1D046099C8D02DBB03CFCE846E32A12A6B8DDF498F8B2F49A0