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DESCRIBE <http://purl.uniprot.org/SHA-384/0A793B3882820672CD4E56DB6F2CA153C8AA2F53E76F87B100367DA761B321C179C2F5B241C35B4B1561CD457C92C0D4>
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http://purl.uniprot.org/SHA-384/0A793B3882820672CD4E56DB6F2CA153C8AA2F53E76F87B100367DA761B321C179C2F5B241C35B4B1561CD457C92C0D4
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/0A793B3882820672CD4E56DB6F2CA153C8AA2F53E76F87B100367DA761B321C179C2F5B241C35B4B1561CD457C92C0D4
http://www.w3.org/2000/01/rdf-schema#comment
"HAGLR aggravates neuropathic pain and promotes inflammatory response and apoptosis of lipopolysaccharide-treated SH-SY5Y cells by sequestering miR-182-5p from ATAT1 and activating NLRP3 inflammasome."
xsd:string
http://purl.uniprot.org/uniprot/#_F3309D7B62CE3F87FEACC27FFCDBB3734055D644B8DE50F648662FD79DA520E525808961F65DD654E59A6DA1F5414876
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/0A793B3882820672CD4E56DB6F2CA153C8AA2F53E76F87B100367DA761B321C179C2F5B241C35B4B1561CD457C92C0D4
http://purl.uniprot.org/uniprot/Q6MG11
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/0A793B3882820672CD4E56DB6F2CA153C8AA2F53E76F87B100367DA761B321C179C2F5B241C35B4B1561CD457C92C0D4
http://purl.uniprot.org/uniprot/#_Q6MG11-mappedCitation-33626373
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/0A793B3882820672CD4E56DB6F2CA153C8AA2F53E76F87B100367DA761B321C179C2F5B241C35B4B1561CD457C92C0D4