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DESCRIBE <http://purl.uniprot.org/SHA-384/0B0568BB07A25C67E0A1A78FA2B9C7DFA7FA6435C7C358D0F20DEA044F6E3278F093CFDA7C28A49306B50C64256C7E7F>
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http://purl.uniprot.org/SHA-384/0B0568BB07A25C67E0A1A78FA2B9C7DFA7FA6435C7C358D0F20DEA044F6E3278F093CFDA7C28A49306B50C64256C7E7F
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/0B0568BB07A25C67E0A1A78FA2B9C7DFA7FA6435C7C358D0F20DEA044F6E3278F093CFDA7C28A49306B50C64256C7E7F
http://www.w3.org/2000/01/rdf-schema#comment
"Ten NPHS2 autosomal recessive mutations have been identified in corticosteroid resistant familial nephrotic syndrome. Podocin plays a major role in glomerular filtration and podocyte physiology."
xsd:string
http://purl.uniprot.org/uniprot/#_1074532E0A43F7B47C4136BCBA6D574743016CDA896364F060B566F52B78156F2C8391E56B3A5BD66633E48E3953E251
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/0B0568BB07A25C67E0A1A78FA2B9C7DFA7FA6435C7C358D0F20DEA044F6E3278F093CFDA7C28A49306B50C64256C7E7F
http://purl.uniprot.org/uniprot/Q9NP85
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/0B0568BB07A25C67E0A1A78FA2B9C7DFA7FA6435C7C358D0F20DEA044F6E3278F093CFDA7C28A49306B50C64256C7E7F
http://purl.uniprot.org/uniprot/#_Q9NP85-mappedCitation-11908478
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/0B0568BB07A25C67E0A1A78FA2B9C7DFA7FA6435C7C358D0F20DEA044F6E3278F093CFDA7C28A49306B50C64256C7E7F