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DESCRIBE <http://purl.uniprot.org/SHA-384/0B4E46D941DB5DD73F5A9C18C0DE5AEA3740E1EAA28E26BB64E7113C7E01F7269642F4DC30FDA5D255C3D1FBA1667043>
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http://purl.uniprot.org/SHA-384/0B4E46D941DB5DD73F5A9C18C0DE5AEA3740E1EAA28E26BB64E7113C7E01F7269642F4DC30FDA5D255C3D1FBA1667043
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/0B4E46D941DB5DD73F5A9C18C0DE5AEA3740E1EAA28E26BB64E7113C7E01F7269642F4DC30FDA5D255C3D1FBA1667043
http://www.w3.org/2000/01/rdf-schema#comment
"Data suggest that activation of cardiac myofilaments is tightly coupled to the open state of the N-domain of cardiac troponin C and that pathological effects of phosphorylation are influenced by mutations in cardiac troponin I."
xsd:string
http://purl.uniprot.org/uniprot/#_A92F79FF798EA7F462D7439B5278D1FCF07B865FA8C75238639000D3A7E0957F2EB500AA5DF35BB37D023AEBA36B3478
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/0B4E46D941DB5DD73F5A9C18C0DE5AEA3740E1EAA28E26BB64E7113C7E01F7269642F4DC30FDA5D255C3D1FBA1667043
http://purl.uniprot.org/uniprot/P63316
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/0B4E46D941DB5DD73F5A9C18C0DE5AEA3740E1EAA28E26BB64E7113C7E01F7269642F4DC30FDA5D255C3D1FBA1667043
http://purl.uniprot.org/uniprot/#_P63316-mappedCitation-15147183
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/0B4E46D941DB5DD73F5A9C18C0DE5AEA3740E1EAA28E26BB64E7113C7E01F7269642F4DC30FDA5D255C3D1FBA1667043