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DESCRIBE <http://purl.uniprot.org/SHA-384/0C362AB29AB6946D5DF3F5881724D7A4945D9B045CBEECA150815CA7198FD8DA5C2A2D15DF3F77D7E32466C4F4069B88>
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http://purl.uniprot.org/SHA-384/0C362AB29AB6946D5DF3F5881724D7A4945D9B045CBEECA150815CA7198FD8DA5C2A2D15DF3F77D7E32466C4F4069B88
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/0C362AB29AB6946D5DF3F5881724D7A4945D9B045CBEECA150815CA7198FD8DA5C2A2D15DF3F77D7E32466C4F4069B88
http://www.w3.org/2000/01/rdf-schema#comment
"These data suggest that M. leprae upregulates IL-1Ra by a TOLLIP-dependent mechanism; inhibition of TOLLIP may decrease an individual's susceptibility to leprosy and offer a novel therapeutic target for IL-1-dependent diseases."
xsd:string
http://purl.uniprot.org/uniprot/#_77B6040820D6996B0D51DFC509B227776E2C2B7FC6A9F0B4D5F5D981D6EA977784A3143024CBF3ACA03B116EA4909E40
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/0C362AB29AB6946D5DF3F5881724D7A4945D9B045CBEECA150815CA7198FD8DA5C2A2D15DF3F77D7E32466C4F4069B88
http://purl.uniprot.org/uniprot/Q9H0E2
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/0C362AB29AB6946D5DF3F5881724D7A4945D9B045CBEECA150815CA7198FD8DA5C2A2D15DF3F77D7E32466C4F4069B88
http://purl.uniprot.org/uniprot/#_Q9H0E2-mappedCitation-26610735
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/0C362AB29AB6946D5DF3F5881724D7A4945D9B045CBEECA150815CA7198FD8DA5C2A2D15DF3F77D7E32466C4F4069B88