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DESCRIBE <http://purl.uniprot.org/SHA-384/0D054CD3333EF9225B3C1F8D90358EE55D99BA44A658D8756DA3806F1FABAA8A347ED281F3F4848B616022A3C780982C>
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http://purl.uniprot.org/SHA-384/0D054CD3333EF9225B3C1F8D90358EE55D99BA44A658D8756DA3806F1FABAA8A347ED281F3F4848B616022A3C780982C
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/0D054CD3333EF9225B3C1F8D90358EE55D99BA44A658D8756DA3806F1FABAA8A347ED281F3F4848B616022A3C780982C
http://www.w3.org/2000/01/rdf-schema#comment
"The C-type lectin receptor Clec1A plays an important role in the development of experimental autoimmune encephalomyelitis by enhancing antigen presenting ability of dendritic cells and inducing inflammatory cytokine IL-17."
xsd:string
http://purl.uniprot.org/uniprot/#_E00E2DCD7AAB48AA70C0C173F5DA03A9CA1468C47FEB6268EAE9A4F8F4E583267E1A676EE2E3CF55894A508C096038BC
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/0D054CD3333EF9225B3C1F8D90358EE55D99BA44A658D8756DA3806F1FABAA8A347ED281F3F4848B616022A3C780982C
http://purl.uniprot.org/uniprot/Q8BWY2
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/0D054CD3333EF9225B3C1F8D90358EE55D99BA44A658D8756DA3806F1FABAA8A347ED281F3F4848B616022A3C780982C
http://purl.uniprot.org/uniprot/#_Q8BWY2-mappedCitation-35135958
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/0D054CD3333EF9225B3C1F8D90358EE55D99BA44A658D8756DA3806F1FABAA8A347ED281F3F4848B616022A3C780982C