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DESCRIBE <http://purl.uniprot.org/SHA-384/0E8C2AB6BEF20E1431AF07F6BF0504BB779E1E6E8633241D8F1D2A8FB1ED578F7192D3975A5FEA417487E8F3654A3082>
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http://purl.uniprot.org/SHA-384/0E8C2AB6BEF20E1431AF07F6BF0504BB779E1E6E8633241D8F1D2A8FB1ED578F7192D3975A5FEA417487E8F3654A3082
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/0E8C2AB6BEF20E1431AF07F6BF0504BB779E1E6E8633241D8F1D2A8FB1ED578F7192D3975A5FEA417487E8F3654A3082
http://www.w3.org/2000/01/rdf-schema#comment
"The protective mechanism exerted by OPC-31260 stems from its influence on the renal vasopressin V(2) receptors. These observations might suggest an effective approach to the treatment of global hypoxia-induced cerebral oedema in humans."
xsd:string
http://purl.uniprot.org/uniprot/#_00F12249E30D60E1C679CE3ADBF71F7DD17182B0BDEA6A0F9DF2CC850D64D354B0E11C80719B8A78325CCA59C96E9181
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/0E8C2AB6BEF20E1431AF07F6BF0504BB779E1E6E8633241D8F1D2A8FB1ED578F7192D3975A5FEA417487E8F3654A3082
http://purl.uniprot.org/uniprot/Q16271
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/0E8C2AB6BEF20E1431AF07F6BF0504BB779E1E6E8633241D8F1D2A8FB1ED578F7192D3975A5FEA417487E8F3654A3082
http://purl.uniprot.org/uniprot/#_Q16271-mappedCitation-18288441
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/0E8C2AB6BEF20E1431AF07F6BF0504BB779E1E6E8633241D8F1D2A8FB1ED578F7192D3975A5FEA417487E8F3654A3082