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DESCRIBE <http://purl.uniprot.org/SHA-384/106FF4BD5ACD0140B3C607B4CAB9E5C927F962433A12926D1947EE142BB6A77ECB345E4C6B0664AC7D389F18979762F7>
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http://purl.uniprot.org/SHA-384/106FF4BD5ACD0140B3C607B4CAB9E5C927F962433A12926D1947EE142BB6A77ECB345E4C6B0664AC7D389F18979762F7
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/106FF4BD5ACD0140B3C607B4CAB9E5C927F962433A12926D1947EE142BB6A77ECB345E4C6B0664AC7D389F18979762F7
http://www.w3.org/2000/01/rdf-schema#comment
"These results suggest that calmodulin plays a crucial role in the processes of Ca(2+)-induced neuronal cell death and the possibility that the blockage of calmodulin attenuates brain injury after cerebral ischemia."
xsd:string
http://purl.uniprot.org/uniprot/#_4AF2DA0168935395F690C2D3AD909AF989E51516F67E54FE4B5CA99AF4FFB09D2D631AEBAFC82062D5D4A0E7960F7ED8
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/106FF4BD5ACD0140B3C607B4CAB9E5C927F962433A12926D1947EE142BB6A77ECB345E4C6B0664AC7D389F18979762F7
http://purl.uniprot.org/uniprot/B4DJ51
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/106FF4BD5ACD0140B3C607B4CAB9E5C927F962433A12926D1947EE142BB6A77ECB345E4C6B0664AC7D389F18979762F7
http://purl.uniprot.org/uniprot/#_B4DJ51-mappedCitation-16545345
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/106FF4BD5ACD0140B3C607B4CAB9E5C927F962433A12926D1947EE142BB6A77ECB345E4C6B0664AC7D389F18979762F7