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DESCRIBE <http://purl.uniprot.org/SHA-384/1070851CC41F35460E6E029889A907E1A93CBF02379158E02C603BC917406AC5337AD242A176BBE3EA71F710BCB6324C>
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http://purl.uniprot.org/SHA-384/1070851CC41F35460E6E029889A907E1A93CBF02379158E02C603BC917406AC5337AD242A176BBE3EA71F710BCB6324C
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/1070851CC41F35460E6E029889A907E1A93CBF02379158E02C603BC917406AC5337AD242A176BBE3EA71F710BCB6324C
http://www.w3.org/2000/01/rdf-schema#comment
"ATP released from BLM-injured lung cells constitutes a major endogenous danger signal that engages the P2X(7) receptor/pannexin-1 axis leading to IL-1beta maturation and lung fibrosis."
xsd:string
http://purl.uniprot.org/uniprot/#_12F2435F8DB971F88537BC7CA1FAC13921DCBB9F55D890B83EDD6DBDBEDE3CD82B172C6D3D3580E28B54F5E1C9276953
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/1070851CC41F35460E6E029889A907E1A93CBF02379158E02C603BC917406AC5337AD242A176BBE3EA71F710BCB6324C
http://purl.uniprot.org/uniprot/Q9Z1M0
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1070851CC41F35460E6E029889A907E1A93CBF02379158E02C603BC917406AC5337AD242A176BBE3EA71F710BCB6324C
http://purl.uniprot.org/uniprot/#_Q9Z1M0-mappedCitation-20522787
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1070851CC41F35460E6E029889A907E1A93CBF02379158E02C603BC917406AC5337AD242A176BBE3EA71F710BCB6324C