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DESCRIBE <http://purl.uniprot.org/SHA-384/11552513434B3F4B23E8476D74BDFB550D68BC191502F0F9CB4B07AFD2B392621DE34EF33E6686CF113D1B1BCB063C0C>
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http://purl.uniprot.org/SHA-384/11552513434B3F4B23E8476D74BDFB550D68BC191502F0F9CB4B07AFD2B392621DE34EF33E6686CF113D1B1BCB063C0C
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/11552513434B3F4B23E8476D74BDFB550D68BC191502F0F9CB4B07AFD2B392621DE34EF33E6686CF113D1B1BCB063C0C
http://www.w3.org/2000/01/rdf-schema#comment
"This study provides a direct demonstration that Tdp1 repairs Topo I covalent lesions in vivo and suggests that spinocerebellar ataxia with axonal neuropathy (SCAN1) arises from the recessive neomorphic mutation H493R."
xsd:string
http://purl.uniprot.org/uniprot/#_7049FF2B2551B8321DCEECECB34BD6F9E8C0F9B3C54E7E2EBEFDCD95F5299CDC09764FBA3156B4FCF40E32EA22291E6F
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/11552513434B3F4B23E8476D74BDFB550D68BC191502F0F9CB4B07AFD2B392621DE34EF33E6686CF113D1B1BCB063C0C
http://purl.uniprot.org/uniprot/Q6P1B8
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/11552513434B3F4B23E8476D74BDFB550D68BC191502F0F9CB4B07AFD2B392621DE34EF33E6686CF113D1B1BCB063C0C
http://purl.uniprot.org/uniprot/#_Q6P1B8-mappedCitation-17948061
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/11552513434B3F4B23E8476D74BDFB550D68BC191502F0F9CB4B07AFD2B392621DE34EF33E6686CF113D1B1BCB063C0C