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http://purl.uniprot.org/SHA-384/12C18ED607327BAB23A215B8C365EB57FB9C8D6AE1970943E1A5C79D5286E3B8CCDBCAEE042E1713ED461053E4575494http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/12C18ED607327BAB23A215B8C365EB57FB9C8D6AE1970943E1A5C79D5286E3B8CCDBCAEE042E1713ED461053E4575494http://www.w3.org/2000/01/rdf-schema#comment"These results suggest that circulating full-length CgA is an important inhibitor of angiogenesis and tumor growth and that cleavage of its C-terminal region markedly reduces its activity. Pathophysiological changes in CgA blood levels and/or its fragmentation might regulate disease progression in cancer patients."xsd:string
http://purl.uniprot.org/uniprot/#_6B6227BF2C4A868B47208F8BECF9E4E5E933ECAC429C9779A8467D93538698D38264A948A8F349A1E8A900C3E2B927C4http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/12C18ED607327BAB23A215B8C365EB57FB9C8D6AE1970943E1A5C79D5286E3B8CCDBCAEE042E1713ED461053E4575494
http://purl.uniprot.org/uniprot/P10645http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/12C18ED607327BAB23A215B8C365EB57FB9C8D6AE1970943E1A5C79D5286E3B8CCDBCAEE042E1713ED461053E4575494
http://purl.uniprot.org/uniprot/#_P10645-mappedCitation-27683038http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/12C18ED607327BAB23A215B8C365EB57FB9C8D6AE1970943E1A5C79D5286E3B8CCDBCAEE042E1713ED461053E4575494