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DESCRIBE <http://purl.uniprot.org/SHA-384/1384C15390B281EEF06FA26E26AA49EF4E37AEDBFD4DBDBD4DD3375F6C913A6B6C4D9773D8CB9BBCE933CA89D04E7779>
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http://purl.uniprot.org/SHA-384/1384C15390B281EEF06FA26E26AA49EF4E37AEDBFD4DBDBD4DD3375F6C913A6B6C4D9773D8CB9BBCE933CA89D04E7779
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/1384C15390B281EEF06FA26E26AA49EF4E37AEDBFD4DBDBD4DD3375F6C913A6B6C4D9773D8CB9BBCE933CA89D04E7779
http://www.w3.org/2000/01/rdf-schema#comment
"Loss of encephalitogenic activity of STAT5-deficient autoreactive CD4+ T cells was independent of IFN-gamma or IL-17 production but was due to the impaired expression of GM-CSF a crucial mediator of T-cell pathogenicity."
xsd:string
http://purl.uniprot.org/uniprot/#_01D3AD631152C74F5997534F064EDE93D3B1A5DBFCD040435C4B3A8D1B32A1944986BCA7B4A4F97538A3F91AAA9F508F
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/1384C15390B281EEF06FA26E26AA49EF4E37AEDBFD4DBDBD4DD3375F6C913A6B6C4D9773D8CB9BBCE933CA89D04E7779
http://purl.uniprot.org/uniprot/Q5SX78
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1384C15390B281EEF06FA26E26AA49EF4E37AEDBFD4DBDBD4DD3375F6C913A6B6C4D9773D8CB9BBCE933CA89D04E7779
http://purl.uniprot.org/uniprot/#_Q5SX78-mappedCitation-25412660
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1384C15390B281EEF06FA26E26AA49EF4E37AEDBFD4DBDBD4DD3375F6C913A6B6C4D9773D8CB9BBCE933CA89D04E7779