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DESCRIBE <http://purl.uniprot.org/SHA-384/147C785B2F3C001DE0465B61F99D6D6D7C717646681127B75D099E4E62E636DB8B9AF1E3B707B1DC4066F9F1F64CD5EB>
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http://purl.uniprot.org/SHA-384/147C785B2F3C001DE0465B61F99D6D6D7C717646681127B75D099E4E62E636DB8B9AF1E3B707B1DC4066F9F1F64CD5EB
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/147C785B2F3C001DE0465B61F99D6D6D7C717646681127B75D099E4E62E636DB8B9AF1E3B707B1DC4066F9F1F64CD5EB
http://www.w3.org/2000/01/rdf-schema#comment
"a point mutation on synaptotagmin located outside of the BoNT/B-binding segment inhibited GT1b binding and blocked GT1b-induced potentiation of BoNT/B binding to synaptotagmin-expressing cells."
xsd:string
http://purl.uniprot.org/uniprot/#_56A53BB9181867CD85990FD3C356C8D87339A12DC20B91485D2200F24DC089711AA9F565BFE9CC23BD466D20CDEC08F8
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/147C785B2F3C001DE0465B61F99D6D6D7C717646681127B75D099E4E62E636DB8B9AF1E3B707B1DC4066F9F1F64CD5EB
http://purl.uniprot.org/uniprot/P21707
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/147C785B2F3C001DE0465B61F99D6D6D7C717646681127B75D099E4E62E636DB8B9AF1E3B707B1DC4066F9F1F64CD5EB
http://purl.uniprot.org/uniprot/#_P21707-mappedCitation-31431523
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/147C785B2F3C001DE0465B61F99D6D6D7C717646681127B75D099E4E62E636DB8B9AF1E3B707B1DC4066F9F1F64CD5EB