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DESCRIBE <http://purl.uniprot.org/SHA-384/148FB8DE97F9290CCA7AE117FA49C763289FA52A9535BD092F39891B23621EBDEAAE226512A12FB733DAA6B5FCDBD36C>
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http://purl.uniprot.org/SHA-384/148FB8DE97F9290CCA7AE117FA49C763289FA52A9535BD092F39891B23621EBDEAAE226512A12FB733DAA6B5FCDBD36C
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/148FB8DE97F9290CCA7AE117FA49C763289FA52A9535BD092F39891B23621EBDEAAE226512A12FB733DAA6B5FCDBD36C
http://www.w3.org/2000/01/rdf-schema#comment
"These data reveal a novel "feed-forward" mechanism induced by NF-kappaB which ensures that acutely synthesized IRF-1 operates in concert with NF-kappaB to amplify the immunoproteasome and antigen-processing functions of CD40."
xsd:string
http://purl.uniprot.org/uniprot/#_08D2D502B9CD4A88189D4F9A3E23EB4E26CF72F600BAD54999FF8433DD425F6264A8631E3CAB57C298E9CA6B86947307
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/148FB8DE97F9290CCA7AE117FA49C763289FA52A9535BD092F39891B23621EBDEAAE226512A12FB733DAA6B5FCDBD36C
http://purl.uniprot.org/uniprot/A0A024RCT1
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/148FB8DE97F9290CCA7AE117FA49C763289FA52A9535BD092F39891B23621EBDEAAE226512A12FB733DAA6B5FCDBD36C
http://purl.uniprot.org/uniprot/#_A0A024RCT1-mappedCitation-18694960
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/148FB8DE97F9290CCA7AE117FA49C763289FA52A9535BD092F39891B23621EBDEAAE226512A12FB733DAA6B5FCDBD36C