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DESCRIBE <http://purl.uniprot.org/SHA-384/16065006624B4C7F6A163252945887A3A4529731245C17B272941EC416C71963CBF477DC88099038C68737B578F0502A>
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http://purl.uniprot.org/SHA-384/16065006624B4C7F6A163252945887A3A4529731245C17B272941EC416C71963CBF477DC88099038C68737B578F0502A
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/16065006624B4C7F6A163252945887A3A4529731245C17B272941EC416C71963CBF477DC88099038C68737B578F0502A
http://www.w3.org/2000/01/rdf-schema#comment
"These results demonstrate that endogenous alpha7 nAChRs mechanisms play an important role in chronic inflammatory and neuropathic pain models."
xsd:string
http://purl.uniprot.org/uniprot/#_3C299175484E2F06E138D2194FCB213D218632DEB2F76AD6796EEFDEB13B169A1104B3B9909C101EC0D8F4D8AABDFEAC
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/16065006624B4C7F6A163252945887A3A4529731245C17B272941EC416C71963CBF477DC88099038C68737B578F0502A
http://purl.uniprot.org/uniprot/Q53YJ9
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/16065006624B4C7F6A163252945887A3A4529731245C17B272941EC416C71963CBF477DC88099038C68737B578F0502A
http://purl.uniprot.org/uniprot/#_Q53YJ9-mappedCitation-23811428
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/16065006624B4C7F6A163252945887A3A4529731245C17B272941EC416C71963CBF477DC88099038C68737B578F0502A