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DESCRIBE <http://purl.uniprot.org/SHA-384/16755C4E2C456CB5F730100796E9AECB2DBFBCCD1EE377461AC38F34B4F9C8CCBD600B1E83B98C0604A442C44AF55850>
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http://purl.uniprot.org/SHA-384/16755C4E2C456CB5F730100796E9AECB2DBFBCCD1EE377461AC38F34B4F9C8CCBD600B1E83B98C0604A442C44AF55850
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/16755C4E2C456CB5F730100796E9AECB2DBFBCCD1EE377461AC38F34B4F9C8CCBD600B1E83B98C0604A442C44AF55850
http://www.w3.org/2000/01/rdf-schema#comment
"Genetic variants of CYP21A2 associated to autoimmune Addison's disease(AAD) are in linkage disequilibrium with the main AAD risk locus HLA-DRB1 and CYP21A2 does not constitute an independent susceptibility locus."
xsd:string
http://purl.uniprot.org/uniprot/#_6B2A7C155C936A177C98BBD8C89137AED9B37ECBB65E4088F8DDAFFF9E25B8062E20378C587A8C1C528CB442D0D8F639
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/16755C4E2C456CB5F730100796E9AECB2DBFBCCD1EE377461AC38F34B4F9C8CCBD600B1E83B98C0604A442C44AF55850
http://purl.uniprot.org/uniprot/I6MHJ8
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/16755C4E2C456CB5F730100796E9AECB2DBFBCCD1EE377461AC38F34B4F9C8CCBD600B1E83B98C0604A442C44AF55850
http://purl.uniprot.org/uniprot/#_I6MHJ8-mappedCitation-25249698
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/16755C4E2C456CB5F730100796E9AECB2DBFBCCD1EE377461AC38F34B4F9C8CCBD600B1E83B98C0604A442C44AF55850