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DESCRIBE <http://purl.uniprot.org/SHA-384/16CCC7EA4655AD693B2069D9D73BFD1B6B1097E7D0D45EA75A127941B389E603E5023660CB1FD8B1415F9ABE2EDD54CD>
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http://purl.uniprot.org/SHA-384/16CCC7EA4655AD693B2069D9D73BFD1B6B1097E7D0D45EA75A127941B389E603E5023660CB1FD8B1415F9ABE2EDD54CD
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/16CCC7EA4655AD693B2069D9D73BFD1B6B1097E7D0D45EA75A127941B389E603E5023660CB1FD8B1415F9ABE2EDD54CD
http://www.w3.org/2000/01/rdf-schema#comment
"Kindlin-3 is essential to activate the beta1 beta2 and beta3 integrin classes and loss of Kindlin-3 function is sufficient to cause a LAD-III-like phenotype in mice"
xsd:string
http://purl.uniprot.org/uniprot/#_AC5AB676BECAF6942C8B77C83BAD90AC3DA767EE29B4734EDBDE6D6E40CB428A9E7DC91021B2E8B30B04E7A46A7DA29E
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/16CCC7EA4655AD693B2069D9D73BFD1B6B1097E7D0D45EA75A127941B389E603E5023660CB1FD8B1415F9ABE2EDD54CD
http://purl.uniprot.org/uniprot/Q86UX7
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/16CCC7EA4655AD693B2069D9D73BFD1B6B1097E7D0D45EA75A127941B389E603E5023660CB1FD8B1415F9ABE2EDD54CD
http://purl.uniprot.org/uniprot/#_Q86UX7-mappedCitation-19234461
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/16CCC7EA4655AD693B2069D9D73BFD1B6B1097E7D0D45EA75A127941B389E603E5023660CB1FD8B1415F9ABE2EDD54CD