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DESCRIBE <http://purl.uniprot.org/SHA-384/16F1CAB4B3A2484A1D8C213CD1B051AB04DA0D20768B075D8325C29D64109C90C37916BE76D0463BF6B5A3E80FE3185D>
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http://purl.uniprot.org/SHA-384/16F1CAB4B3A2484A1D8C213CD1B051AB04DA0D20768B075D8325C29D64109C90C37916BE76D0463BF6B5A3E80FE3185D
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/16F1CAB4B3A2484A1D8C213CD1B051AB04DA0D20768B075D8325C29D64109C90C37916BE76D0463BF6B5A3E80FE3185D
http://www.w3.org/2000/01/rdf-schema#comment
"These results suggest that a circulating factor generated after disruption of hepatic Gcgr signaling can increase alpha-cell proliferation independent of direct pancreatic input."
xsd:string
http://purl.uniprot.org/uniprot/#_3F3399149E140FBC107D078F35A56F5413BEF7149A44CF2D688EC55151D7F04543D509D41EAC481D527245A604CAA0F0
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/16F1CAB4B3A2484A1D8C213CD1B051AB04DA0D20768B075D8325C29D64109C90C37916BE76D0463BF6B5A3E80FE3185D
http://purl.uniprot.org/uniprot/Q3UN81
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/16F1CAB4B3A2484A1D8C213CD1B051AB04DA0D20768B075D8325C29D64109C90C37916BE76D0463BF6B5A3E80FE3185D
http://purl.uniprot.org/uniprot/#_Q3UN81-mappedCitation-23160527
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/16F1CAB4B3A2484A1D8C213CD1B051AB04DA0D20768B075D8325C29D64109C90C37916BE76D0463BF6B5A3E80FE3185D