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DESCRIBE <http://purl.uniprot.org/SHA-384/197A09FAE33A182C1C13D58AD9318DA5C7545B7CBA1A03752862CFED3A4572A0BCDE08DCAD40E98D43B4DC513ABC365B>
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http://purl.uniprot.org/SHA-384/197A09FAE33A182C1C13D58AD9318DA5C7545B7CBA1A03752862CFED3A4572A0BCDE08DCAD40E98D43B4DC513ABC365B
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/197A09FAE33A182C1C13D58AD9318DA5C7545B7CBA1A03752862CFED3A4572A0BCDE08DCAD40E98D43B4DC513ABC365B
http://www.w3.org/2000/01/rdf-schema#comment
"Both EMAP II and CXCR3 were essentially required for gp120-induced lung microvascular endothelial cell apoptosis."
xsd:string
http://purl.uniprot.org/uniprot/#_CA2EF1293F44EEBF17EE4E131CD3B1D8856F2B7F91373CFEE7E80F4D4654A1F5F76BA4B27188AA8789EACC97FDBFB54A
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/197A09FAE33A182C1C13D58AD9318DA5C7545B7CBA1A03752862CFED3A4572A0BCDE08DCAD40E98D43B4DC513ABC365B
http://purl.uniprot.org/uniprot/O95834
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/197A09FAE33A182C1C13D58AD9318DA5C7545B7CBA1A03752862CFED3A4572A0BCDE08DCAD40E98D43B4DC513ABC365B
http://purl.uniprot.org/uniprot/#_O95834-mappedCitation-24318111
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/197A09FAE33A182C1C13D58AD9318DA5C7545B7CBA1A03752862CFED3A4572A0BCDE08DCAD40E98D43B4DC513ABC365B