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DESCRIBE <http://purl.uniprot.org/SHA-384/1A785683B258AA785055786AA71978B4B4A9E1D1E0F43D91D304E3CB1B46EF6D46D3F6B83F49A9E29A16F617B4774D8C>
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http://purl.uniprot.org/SHA-384/1A785683B258AA785055786AA71978B4B4A9E1D1E0F43D91D304E3CB1B46EF6D46D3F6B83F49A9E29A16F617B4774D8C
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/1A785683B258AA785055786AA71978B4B4A9E1D1E0F43D91D304E3CB1B46EF6D46D3F6B83F49A9E29A16F617B4774D8C
http://www.w3.org/2000/01/rdf-schema#comment
"The DEGs such as AGTR1 CYP3A4 and CYP4A11 may play critical roles in the development of HTN likely via the regulation by hsa-miR-26b-5p and taking part in some pathways."
xsd:string
http://purl.uniprot.org/uniprot/#_CD83A82D5E74A8DD1B0967E8935C8373BD86F3A73B5D9AA4BBADC4EE2702DA7080C47BA826672FAFB7F8A7CBF448D8D1
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/1A785683B258AA785055786AA71978B4B4A9E1D1E0F43D91D304E3CB1B46EF6D46D3F6B83F49A9E29A16F617B4774D8C
http://purl.uniprot.org/uniprot/A0A0A0MSE3
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1A785683B258AA785055786AA71978B4B4A9E1D1E0F43D91D304E3CB1B46EF6D46D3F6B83F49A9E29A16F617B4774D8C
http://purl.uniprot.org/uniprot/#_A0A0A0MSE3-mappedCitation-27756246
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1A785683B258AA785055786AA71978B4B4A9E1D1E0F43D91D304E3CB1B46EF6D46D3F6B83F49A9E29A16F617B4774D8C