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DESCRIBE <http://purl.uniprot.org/SHA-384/1AE10AD779F0DAE8ED4A27BF7BDCCFEA94D07FA22B778C3F6D26D755D386E1C7E856F4D6A111337120B27D9A93EB011F>
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http://purl.uniprot.org/SHA-384/1AE10AD779F0DAE8ED4A27BF7BDCCFEA94D07FA22B778C3F6D26D755D386E1C7E856F4D6A111337120B27D9A93EB011F
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/1AE10AD779F0DAE8ED4A27BF7BDCCFEA94D07FA22B778C3F6D26D755D386E1C7E856F4D6A111337120B27D9A93EB011F
http://www.w3.org/2000/01/rdf-schema#comment
"These results suggest that during inflammation sTLT-1 may mediate hemostasis by enhancing actin polymerization resulting in increased platelet aggregation and adherence to the endothelium."
xsd:string
http://purl.uniprot.org/uniprot/#_12E652C042AAD4989119CA9987B6F764AA723712CFA11213977118D41F35816D6B79B0A41E946052D9C682CE2D1412EE
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/1AE10AD779F0DAE8ED4A27BF7BDCCFEA94D07FA22B778C3F6D26D755D386E1C7E856F4D6A111337120B27D9A93EB011F
http://purl.uniprot.org/uniprot/Q86YW5
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1AE10AD779F0DAE8ED4A27BF7BDCCFEA94D07FA22B778C3F6D26D755D386E1C7E856F4D6A111337120B27D9A93EB011F
http://purl.uniprot.org/uniprot/#_Q86YW5-mappedCitation-20093931
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1AE10AD779F0DAE8ED4A27BF7BDCCFEA94D07FA22B778C3F6D26D755D386E1C7E856F4D6A111337120B27D9A93EB011F