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DESCRIBE <http://purl.uniprot.org/SHA-384/1BC7A8C257442B3DF4EF5385951764DBB339C6242ABBFB4D2B02D9943468D37822936B810239FFA8807784741F328855>
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http://purl.uniprot.org/SHA-384/1BC7A8C257442B3DF4EF5385951764DBB339C6242ABBFB4D2B02D9943468D37822936B810239FFA8807784741F328855
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/1BC7A8C257442B3DF4EF5385951764DBB339C6242ABBFB4D2B02D9943468D37822936B810239FFA8807784741F328855
http://www.w3.org/2000/01/rdf-schema#comment
"These studies reveal a SHP-1-mediated antiangiogenic pathway induced by CD36-TSP-1 interaction that inhibits VEGFR2 signaling and they provide a novel target to modulate angiogenesis therapeutically."
xsd:string
http://purl.uniprot.org/uniprot/#_B2C072FFB2F6B4C8DEFFE87D71C602754A01041170CBED1D84A1694DD4A2DEEE711080F4A451091949E6F4F227464B14
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/1BC7A8C257442B3DF4EF5385951764DBB339C6242ABBFB4D2B02D9943468D37822936B810239FFA8807784741F328855
http://purl.uniprot.org/uniprot/Q3TPD3
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1BC7A8C257442B3DF4EF5385951764DBB339C6242ABBFB4D2B02D9943468D37822936B810239FFA8807784741F328855
http://purl.uniprot.org/uniprot/#_Q3TPD3-mappedCitation-23896411
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1BC7A8C257442B3DF4EF5385951764DBB339C6242ABBFB4D2B02D9943468D37822936B810239FFA8807784741F328855