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DESCRIBE <http://purl.uniprot.org/SHA-384/1C066CF47E784C710341AA8DB05FEE817DF9B2C9A716BD9AAADC3442E3BC35CA94C56B729C59FD77D137CD6C3201BC6A>
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http://purl.uniprot.org/SHA-384/1C066CF47E784C710341AA8DB05FEE817DF9B2C9A716BD9AAADC3442E3BC35CA94C56B729C59FD77D137CD6C3201BC6A
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/1C066CF47E784C710341AA8DB05FEE817DF9B2C9A716BD9AAADC3442E3BC35CA94C56B729C59FD77D137CD6C3201BC6A
http://www.w3.org/2000/01/rdf-schema#comment
"These results might suggest opposing effects of the HLA-B*40 in the genetic susceptibility to develop ALL or AML and offer the possibility to study further the molecular mechanisms of cell differentiation within the bone marrow lineage."
xsd:string
http://purl.uniprot.org/uniprot/#_61769EC493CC03A892C133E5642B485C0B5A3644C9132EAD3451869B872B419ABCFA5AF5AED877C5C660A81776E689C7
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/1C066CF47E784C710341AA8DB05FEE817DF9B2C9A716BD9AAADC3442E3BC35CA94C56B729C59FD77D137CD6C3201BC6A
http://purl.uniprot.org/uniprot/B2G3G5
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1C066CF47E784C710341AA8DB05FEE817DF9B2C9A716BD9AAADC3442E3BC35CA94C56B729C59FD77D137CD6C3201BC6A
http://purl.uniprot.org/uniprot/#_B2G3G5-mappedCitation-24364037
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1C066CF47E784C710341AA8DB05FEE817DF9B2C9A716BD9AAADC3442E3BC35CA94C56B729C59FD77D137CD6C3201BC6A