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DESCRIBE <http://purl.uniprot.org/SHA-384/1C0B2455C179B3108A921BFF7D6AF088E4F42E824F77661765D4E502BE2A135107635B33A8A131D47D7A591DDF6572C4>
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http://purl.uniprot.org/SHA-384/1C0B2455C179B3108A921BFF7D6AF088E4F42E824F77661765D4E502BE2A135107635B33A8A131D47D7A591DDF6572C4
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/1C0B2455C179B3108A921BFF7D6AF088E4F42E824F77661765D4E502BE2A135107635B33A8A131D47D7A591DDF6572C4
http://www.w3.org/2000/01/rdf-schema#comment
"The authors analyzed the rpn11-m1 phenotypes and found that overexpression of Cdc14 suppresses all the rpn11-m1 defects including the mitochondrial ones."
xsd:string
http://purl.uniprot.org/uniprot/#_6D3EBD77D02594107391CA5ADB17493EAAFED8A5BC8E39F258844D5115C5F905B88520A4BEA572777CD2586EB63FBE8B
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/1C0B2455C179B3108A921BFF7D6AF088E4F42E824F77661765D4E502BE2A135107635B33A8A131D47D7A591DDF6572C4
http://purl.uniprot.org/uniprot/Q00684
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1C0B2455C179B3108A921BFF7D6AF088E4F42E824F77661765D4E502BE2A135107635B33A8A131D47D7A591DDF6572C4
http://purl.uniprot.org/uniprot/#_Q00684-mappedCitation-21059189
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1C0B2455C179B3108A921BFF7D6AF088E4F42E824F77661765D4E502BE2A135107635B33A8A131D47D7A591DDF6572C4