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DESCRIBE <http://purl.uniprot.org/SHA-384/1CCCCBC91DADF68C8773D80C183063CAE76FBD49FAC84362575D6C94CE85DB6FC52DF4A0B452A3C533DA391F0E88E98B>
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http://purl.uniprot.org/SHA-384/1CCCCBC91DADF68C8773D80C183063CAE76FBD49FAC84362575D6C94CE85DB6FC52DF4A0B452A3C533DA391F0E88E98B
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/1CCCCBC91DADF68C8773D80C183063CAE76FBD49FAC84362575D6C94CE85DB6FC52DF4A0B452A3C533DA391F0E88E98B
http://www.w3.org/2000/01/rdf-schema#comment
"Results indicate that up-regulation of anionic trypsinogen in pancreatic diseases does not affect physiological trypsinogen activation but significantly limits trypsin generation under potential pathological conditions."
xsd:string
http://purl.uniprot.org/uniprot/#_E2BF1D3296248F1044B8A0D9D72C71953F1B9861CFCB1E8890CBDC8ABC561DBFA9F01A52A05541AC481063919037F93B
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/1CCCCBC91DADF68C8773D80C183063CAE76FBD49FAC84362575D6C94CE85DB6FC52DF4A0B452A3C533DA391F0E88E98B
http://purl.uniprot.org/uniprot/Q5NV56
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1CCCCBC91DADF68C8773D80C183063CAE76FBD49FAC84362575D6C94CE85DB6FC52DF4A0B452A3C533DA391F0E88E98B
http://purl.uniprot.org/uniprot/#_Q5NV56-mappedCitation-12709065
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1CCCCBC91DADF68C8773D80C183063CAE76FBD49FAC84362575D6C94CE85DB6FC52DF4A0B452A3C533DA391F0E88E98B