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DESCRIBE <http://purl.uniprot.org/SHA-384/1DE8E348238EF5ED2D7F12D48631F7C344A62A337758DFB248D8B799ED479CC64E386400438B67D5FE2B4328EBBC5B93>
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http://purl.uniprot.org/SHA-384/1DE8E348238EF5ED2D7F12D48631F7C344A62A337758DFB248D8B799ED479CC64E386400438B67D5FE2B4328EBBC5B93
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/1DE8E348238EF5ED2D7F12D48631F7C344A62A337758DFB248D8B799ED479CC64E386400438B67D5FE2B4328EBBC5B93
http://www.w3.org/2000/01/rdf-schema#comment
"Findings suggest that TRPC6-dependent Ca(2+) entry into endothelial cells secondary to TLR4-induced diacylglycerol generation participates in mediating both lung vascular barrier disruption and inflammation induced by endotoxin."
xsd:string
http://purl.uniprot.org/uniprot/#_C8182224D762A12F59D65F1BEBA166495B153C50748B14DE294A58CF85E9DD7B71F6E906B3EDA167595837E96BCC5F54
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/1DE8E348238EF5ED2D7F12D48631F7C344A62A337758DFB248D8B799ED479CC64E386400438B67D5FE2B4328EBBC5B93
http://purl.uniprot.org/uniprot/Q3U4D4
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1DE8E348238EF5ED2D7F12D48631F7C344A62A337758DFB248D8B799ED479CC64E386400438B67D5FE2B4328EBBC5B93
http://purl.uniprot.org/uniprot/#_Q3U4D4-mappedCitation-23045603
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1DE8E348238EF5ED2D7F12D48631F7C344A62A337758DFB248D8B799ED479CC64E386400438B67D5FE2B4328EBBC5B93