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DESCRIBE <http://purl.uniprot.org/SHA-384/1E10D84B2D445E1E1C866860D1511C4AD8939DB25D79E36786FDA6D161364484A412103056630ECDD88C0FAA8EA0F8DD>
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http://purl.uniprot.org/SHA-384/1E10D84B2D445E1E1C866860D1511C4AD8939DB25D79E36786FDA6D161364484A412103056630ECDD88C0FAA8EA0F8DD
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/1E10D84B2D445E1E1C866860D1511C4AD8939DB25D79E36786FDA6D161364484A412103056630ECDD88C0FAA8EA0F8DD
http://www.w3.org/2000/01/rdf-schema#comment
"neither ALK5 nor TGFBR2 is required for ALK1 signaling pertinent to the pathogenesis of HHT and suggest that HHT might not be a TGF-beta subfamily disease."
xsd:string
http://purl.uniprot.org/uniprot/#_F19DEC2E9E0EC0D96B7B1E9E9A5315B5F4F88F3EF01FF31CF3CDA51E9CB61EAE0754D82345D4AB6B1BF79C7ED7EC298C
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/1E10D84B2D445E1E1C866860D1511C4AD8939DB25D79E36786FDA6D161364484A412103056630ECDD88C0FAA8EA0F8DD
http://purl.uniprot.org/uniprot/Q61288
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1E10D84B2D445E1E1C866860D1511C4AD8939DB25D79E36786FDA6D161364484A412103056630ECDD88C0FAA8EA0F8DD
http://purl.uniprot.org/uniprot/#_Q61288-mappedCitation-17911384
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/1E10D84B2D445E1E1C866860D1511C4AD8939DB25D79E36786FDA6D161364484A412103056630ECDD88C0FAA8EA0F8DD