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http://purl.uniprot.org/SHA-384/1F570EB527A0ACC1B74C3BF273E6BE9F9E75F6860A9C1C75A39F084185B7696F5F7EC497FD8EE1788A669E0F56546F47http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/1F570EB527A0ACC1B74C3BF273E6BE9F9E75F6860A9C1C75A39F084185B7696F5F7EC497FD8EE1788A669E0F56546F47http://www.w3.org/2000/01/rdf-schema#comment"Ibrutinib-resistant TMD8 cells had higher BCL2 gene expression and increased sensitivity to ABT-199 a BCL-2 inhibitor. Consistently clinical samples from ABC-DLBCL patients who experienced poorer response to ibrutinib had higher BCL2 gene expression. We further demonstrated synergistic growth suppression by ibrutinib and ABT-199 in multiple ABC-DLBCL GCB-DLBCL and follicular lymphoma cell lines."xsd:string
http://purl.uniprot.org/uniprot/#_6881E090CDAA74C07ABF7BE303A58DF16928AA2D0259F7BF88DB06D1467E90E3B91703162189518B913010C10DCF16D5http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/1F570EB527A0ACC1B74C3BF273E6BE9F9E75F6860A9C1C75A39F084185B7696F5F7EC497FD8EE1788A669E0F56546F47
http://purl.uniprot.org/uniprot/Q572P3http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/1F570EB527A0ACC1B74C3BF273E6BE9F9E75F6860A9C1C75A39F084185B7696F5F7EC497FD8EE1788A669E0F56546F47
http://purl.uniprot.org/uniprot/#_Q572P3-mappedCitation-28428442http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/1F570EB527A0ACC1B74C3BF273E6BE9F9E75F6860A9C1C75A39F084185B7696F5F7EC497FD8EE1788A669E0F56546F47