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DESCRIBE <http://purl.uniprot.org/SHA-384/20CF7FBC0E3E5BA6E89EFA8AE42BED795ECEEFE10F0716AB66B75D4AD8967788C3AD11491BB5F7B22FCCB4B1E4B05560>
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http://purl.uniprot.org/SHA-384/20CF7FBC0E3E5BA6E89EFA8AE42BED795ECEEFE10F0716AB66B75D4AD8967788C3AD11491BB5F7B22FCCB4B1E4B05560
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/20CF7FBC0E3E5BA6E89EFA8AE42BED795ECEEFE10F0716AB66B75D4AD8967788C3AD11491BB5F7B22FCCB4B1E4B05560
http://www.w3.org/2000/01/rdf-schema#comment
"Microvascular endothelial cells are more susceptible than aortic cells to advanced glycation end products-enhanced permeability and that AGE-enhanced permeability is dependent on VEGF expression induced by reactive oxygen species."
xsd:string
http://purl.uniprot.org/uniprot/#_58E8E546B09BD7CC7385D21D87AC113FFC06D6AC9D6AE692CF9D408A37047A6A6C16B2561FC390C60F94734B5C64C486
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/20CF7FBC0E3E5BA6E89EFA8AE42BED795ECEEFE10F0716AB66B75D4AD8967788C3AD11491BB5F7B22FCCB4B1E4B05560
http://purl.uniprot.org/uniprot/C6KYY4
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/20CF7FBC0E3E5BA6E89EFA8AE42BED795ECEEFE10F0716AB66B75D4AD8967788C3AD11491BB5F7B22FCCB4B1E4B05560
http://purl.uniprot.org/uniprot/#_C6KYY4-mappedCitation-21296593
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/20CF7FBC0E3E5BA6E89EFA8AE42BED795ECEEFE10F0716AB66B75D4AD8967788C3AD11491BB5F7B22FCCB4B1E4B05560