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DESCRIBE <http://purl.uniprot.org/SHA-384/22EBF01DF5F9D3869A272927637C20B5BE892ADA58F823306F4DDBE57F0F72156E74B2BBF167E65435C95BC4BE958A20>
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http://purl.uniprot.org/SHA-384/22EBF01DF5F9D3869A272927637C20B5BE892ADA58F823306F4DDBE57F0F72156E74B2BBF167E65435C95BC4BE958A20
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/22EBF01DF5F9D3869A272927637C20B5BE892ADA58F823306F4DDBE57F0F72156E74B2BBF167E65435C95BC4BE958A20
http://www.w3.org/2000/01/rdf-schema#comment
"Data show that peptidyl-prolyl isomerase (Pin1) inhibition inhibits high glucose (HG)-induced cardiac fibroblasts (CFs) proliferation and migration."
xsd:string
http://purl.uniprot.org/uniprot/#_2F5C911AB3EBCE3CDC88F613F14AAD35B401EC6BDC2C3DD65755233A1B7BF53A75B3D48CFC9763800F0944E14AE0C068
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/22EBF01DF5F9D3869A272927637C20B5BE892ADA58F823306F4DDBE57F0F72156E74B2BBF167E65435C95BC4BE958A20
http://purl.uniprot.org/uniprot/Q3UTI7
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/22EBF01DF5F9D3869A272927637C20B5BE892ADA58F823306F4DDBE57F0F72156E74B2BBF167E65435C95BC4BE958A20
http://purl.uniprot.org/uniprot/#_Q3UTI7-mappedCitation-27634219
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/22EBF01DF5F9D3869A272927637C20B5BE892ADA58F823306F4DDBE57F0F72156E74B2BBF167E65435C95BC4BE958A20