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http://purl.uniprot.org/SHA-384/237258C722DB5B06B3B13A0131E48A11C4E5F1851F0ED4EEDC9A5A2B11F72B0F2B346ACA73B2C55BB88D5E3E975CC691http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/237258C722DB5B06B3B13A0131E48A11C4E5F1851F0ED4EEDC9A5A2B11F72B0F2B346ACA73B2C55BB88D5E3E975CC691http://www.w3.org/2000/01/rdf-schema#comment"Conversely treatment with LY294002 (a selective inhibitor of Akt1) reversed the effects of quercetin. In conclusion these findings highlight the important role of quercetin in protecting against cognitive deficits and inhibiting neuronal apoptosis via the Akt signaling pathway. We believe that quercetin might prove to be a useful therapeutic component in treating cerebral I/R diseases in the near future."xsd:string
http://purl.uniprot.org/uniprot/#_8E7349F49A883BCF65BF7E72E07CFBF0A4D3AC400E8F04E69119EC2F4282BF5769FAB08DA0C362E3BBED68E5FFC05ACBhttp://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/237258C722DB5B06B3B13A0131E48A11C4E5F1851F0ED4EEDC9A5A2B11F72B0F2B346ACA73B2C55BB88D5E3E975CC691
http://purl.uniprot.org/uniprot/Q78GB8http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/237258C722DB5B06B3B13A0131E48A11C4E5F1851F0ED4EEDC9A5A2B11F72B0F2B346ACA73B2C55BB88D5E3E975CC691
http://purl.uniprot.org/uniprot/#_Q78GB8-mappedCitation-27450812http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/237258C722DB5B06B3B13A0131E48A11C4E5F1851F0ED4EEDC9A5A2B11F72B0F2B346ACA73B2C55BB88D5E3E975CC691