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DESCRIBE <http://purl.uniprot.org/SHA-384/2422D93D6BEC9619DFA1613C261B056B321AE89435AF1CD1D5B5E2FC4F2B42F2EEB8512C8C67C933A9DFE641101E79CA>
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http://purl.uniprot.org/SHA-384/2422D93D6BEC9619DFA1613C261B056B321AE89435AF1CD1D5B5E2FC4F2B42F2EEB8512C8C67C933A9DFE641101E79CA
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/2422D93D6BEC9619DFA1613C261B056B321AE89435AF1CD1D5B5E2FC4F2B42F2EEB8512C8C67C933A9DFE641101E79CA
http://www.w3.org/2000/01/rdf-schema#comment
"Apaf-1(-/-) (null) fibroblasts were resistant to cell death initiated by proapoptotic stimuli such as tamoxifen; susceptible to apoptosis induced by thapsigargin and brefeldin-A both of which induce ER stress"
xsd:string
http://purl.uniprot.org/uniprot/#_36993B0357BB2F45040E70BCC3246D4F1DF2F50C878CBC27FA65C799B13ECF62DC14E7E3A13520FA9C4C28F1FCB3F7E3
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/2422D93D6BEC9619DFA1613C261B056B321AE89435AF1CD1D5B5E2FC4F2B42F2EEB8512C8C67C933A9DFE641101E79CA
http://purl.uniprot.org/uniprot/G3XA09
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/2422D93D6BEC9619DFA1613C261B056B321AE89435AF1CD1D5B5E2FC4F2B42F2EEB8512C8C67C933A9DFE641101E79CA
http://purl.uniprot.org/uniprot/#_G3XA09-mappedCitation-11919205
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/2422D93D6BEC9619DFA1613C261B056B321AE89435AF1CD1D5B5E2FC4F2B42F2EEB8512C8C67C933A9DFE641101E79CA