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http://purl.uniprot.org/SHA-384/27340C715F9FD11E0B47C663FA46E46AE17FA284F796F7916B41FEDB0C014501DBA13B06A17E8733E3FF81A22C2F1CABhttp://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/27340C715F9FD11E0B47C663FA46E46AE17FA284F796F7916B41FEDB0C014501DBA13B06A17E8733E3FF81A22C2F1CABhttp://www.w3.org/2000/01/rdf-schema#comment"These observations demonstrate that activation of the renin-angiotensin system (RAS) promotes colitis in a blood pressure independent manner. Angiotensin II appears to drive colonic mucosal inflammation by promoting intestinal epithelial cell apoptosis and mucosal TH17 responses in colitis development"xsd:string
http://purl.uniprot.org/uniprot/#_A030016B29E4250832B30F8BEA5367B133AADF0038E24F1A9CEB9C02C859F6243A93F6B18F11E3D3CBEFA0E8159D98A5http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/27340C715F9FD11E0B47C663FA46E46AE17FA284F796F7916B41FEDB0C014501DBA13B06A17E8733E3FF81A22C2F1CAB
http://purl.uniprot.org/uniprot/Q9UQK5http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/27340C715F9FD11E0B47C663FA46E46AE17FA284F796F7916B41FEDB0C014501DBA13B06A17E8733E3FF81A22C2F1CAB
http://purl.uniprot.org/uniprot/#_Q9UQK5-mappedCitation-27271344http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/27340C715F9FD11E0B47C663FA46E46AE17FA284F796F7916B41FEDB0C014501DBA13B06A17E8733E3FF81A22C2F1CAB