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DESCRIBE <http://purl.uniprot.org/SHA-384/28B0CB3ED1A30F19371BDA6241406785028FD9C2B39CE9D923206DC252A9BBA0699C9E71D077D3812155F96ACD27E465>
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http://purl.uniprot.org/SHA-384/28B0CB3ED1A30F19371BDA6241406785028FD9C2B39CE9D923206DC252A9BBA0699C9E71D077D3812155F96ACD27E465
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/28B0CB3ED1A30F19371BDA6241406785028FD9C2B39CE9D923206DC252A9BBA0699C9E71D077D3812155F96ACD27E465
http://www.w3.org/2000/01/rdf-schema#comment
"the beneficial actions of insulin in diabetic nephropathy appear to be mediated in part by suppressing renal Nrf2 and Agt gene transcription and preventing Nrf2 stimulation of Agt expression via hnRNP F/K."
xsd:string
http://purl.uniprot.org/uniprot/#_0938815A9D873E39A33A89D716015A1168BFA12D7F3F87A1B7B7B8F6F055DE27C3EB1A97000C5F36F7231292BEA3A800
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/28B0CB3ED1A30F19371BDA6241406785028FD9C2B39CE9D923206DC252A9BBA0699C9E71D077D3812155F96ACD27E465
http://purl.uniprot.org/uniprot/Q3U9Q3
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/28B0CB3ED1A30F19371BDA6241406785028FD9C2B39CE9D923206DC252A9BBA0699C9E71D077D3812155F96ACD27E465
http://purl.uniprot.org/uniprot/#_Q3U9Q3-mappedCitation-28324005
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/28B0CB3ED1A30F19371BDA6241406785028FD9C2B39CE9D923206DC252A9BBA0699C9E71D077D3812155F96ACD27E465