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DESCRIBE <http://purl.uniprot.org/SHA-384/2A604216146A3A8A53A444708B990C9CE9DCCEFF0CA2A1F2854050B36A1DBB1A12C74F90A680BF273E7DA72AF11E8575>
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http://purl.uniprot.org/SHA-384/2A604216146A3A8A53A444708B990C9CE9DCCEFF0CA2A1F2854050B36A1DBB1A12C74F90A680BF273E7DA72AF11E8575
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/2A604216146A3A8A53A444708B990C9CE9DCCEFF0CA2A1F2854050B36A1DBB1A12C74F90A680BF273E7DA72AF11E8575
http://www.w3.org/2000/01/rdf-schema#comment
"these data implicate an important role for TIEG1 in mediating estrogen signaling throughout the mouse skeleton and suggest that defects in this pathway are likely to contribute to the sex-specific osteopenic phenotype"
xsd:string
http://purl.uniprot.org/uniprot/#_024EFFE079D1F215A2AEFB64BFDAA0E79005188983F92B0E9A3E3DD9FD2701FEC7442B7676CADFE31205CB3685A0FCBF
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/2A604216146A3A8A53A444708B990C9CE9DCCEFF0CA2A1F2854050B36A1DBB1A12C74F90A680BF273E7DA72AF11E8575
http://purl.uniprot.org/uniprot/Q61596
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/2A604216146A3A8A53A444708B990C9CE9DCCEFF0CA2A1F2854050B36A1DBB1A12C74F90A680BF273E7DA72AF11E8575
http://purl.uniprot.org/uniprot/#_Q61596-mappedCitation-24190163
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/2A604216146A3A8A53A444708B990C9CE9DCCEFF0CA2A1F2854050B36A1DBB1A12C74F90A680BF273E7DA72AF11E8575