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DESCRIBE <http://purl.uniprot.org/SHA-384/2B3C481CF966C59EBD9154B934B1783D04D7F7F2DB56D695C0D3B016BAE227A47426D02F697495B7A4EB4D46E0BA5CED>
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http://purl.uniprot.org/SHA-384/2B3C481CF966C59EBD9154B934B1783D04D7F7F2DB56D695C0D3B016BAE227A47426D02F697495B7A4EB4D46E0BA5CED
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/2B3C481CF966C59EBD9154B934B1783D04D7F7F2DB56D695C0D3B016BAE227A47426D02F697495B7A4EB4D46E0BA5CED
http://www.w3.org/2000/01/rdf-schema#comment
"Increased miR-155 relieves chronic inflammation by a negative feedback loop and plays a protective role during atherosclerosis-associated foam cell formation by signaling through the miR-155-CARHSP1-TNF-alpha pathway."
xsd:string
http://purl.uniprot.org/uniprot/#_09A2CE25D462CBB23B98253C5685905452414F538A2962BC7A1C65635303E48BE4A291F79D9C5A48145A9E3D963AC069
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/2B3C481CF966C59EBD9154B934B1783D04D7F7F2DB56D695C0D3B016BAE227A47426D02F697495B7A4EB4D46E0BA5CED
http://purl.uniprot.org/uniprot/Q9Y2V2
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/2B3C481CF966C59EBD9154B934B1783D04D7F7F2DB56D695C0D3B016BAE227A47426D02F697495B7A4EB4D46E0BA5CED
http://purl.uniprot.org/uniprot/#_Q9Y2V2-mappedCitation-26899994
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/2B3C481CF966C59EBD9154B934B1783D04D7F7F2DB56D695C0D3B016BAE227A47426D02F697495B7A4EB4D46E0BA5CED