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DESCRIBE <http://purl.uniprot.org/SHA-384/2B4B40160F2E4B8757EE5246227DB34B2BF3108645EDC06F20E1D812845A16A842BFFDC148C28C21B5F5AF228E77B21E>
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http://purl.uniprot.org/SHA-384/2B4B40160F2E4B8757EE5246227DB34B2BF3108645EDC06F20E1D812845A16A842BFFDC148C28C21B5F5AF228E77B21E
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/2B4B40160F2E4B8757EE5246227DB34B2BF3108645EDC06F20E1D812845A16A842BFFDC148C28C21B5F5AF228E77B21E
http://www.w3.org/2000/01/rdf-schema#comment
"The acceleration of lesion development in the absence of DAF provides confirmation of the proinflammatory and proatherosclerotic potential of complement activation in the Ldlr(-/-) mouse model."
xsd:string
http://purl.uniprot.org/uniprot/#_CA3E4A2C4FD7F96BEB29114C7EE2FABAD566C98090DB088B2C2AEE5E83E040164CB34D08028A3167971416D64A1A0DF5
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/2B4B40160F2E4B8757EE5246227DB34B2BF3108645EDC06F20E1D812845A16A842BFFDC148C28C21B5F5AF228E77B21E
http://purl.uniprot.org/uniprot/Q61475
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/2B4B40160F2E4B8757EE5246227DB34B2BF3108645EDC06F20E1D812845A16A842BFFDC148C28C21B5F5AF228E77B21E
http://purl.uniprot.org/uniprot/#_Q61475-mappedCitation-19729477
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/2B4B40160F2E4B8757EE5246227DB34B2BF3108645EDC06F20E1D812845A16A842BFFDC148C28C21B5F5AF228E77B21E