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http://purl.uniprot.org/SHA-384/2B9E0E0730A10F9D7C199D3C6F674C72E5581DE09D3A995B52C34A56D4CEA55B14683D5072B5FB3F4CA15CCADA0F7961http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/2B9E0E0730A10F9D7C199D3C6F674C72E5581DE09D3A995B52C34A56D4CEA55B14683D5072B5FB3F4CA15CCADA0F7961http://www.w3.org/2000/01/rdf-schema#comment"Silencing of KIF3A suppressed TNBC cell proliferation by repressing the Rb-E2F signaling pathway and inhibited migration and invasion by repressing epithelial-mesenchymal transition. The tumor size was smaller and the number of lung metastatic nodules was lower in KIF3A depletion MDA-MB-231 cell xenograft mice than in the negative control group. In addition KIF3A overexpression correlated with chemoresistance."xsd:string
http://purl.uniprot.org/uniprot/#_927A01CB4D07D502104CD0184D534C0C7A767675892152B495E6C6DDD933423E80BE0B5ED038CC627DFCA26343F2846Bhttp://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/2B9E0E0730A10F9D7C199D3C6F674C72E5581DE09D3A995B52C34A56D4CEA55B14683D5072B5FB3F4CA15CCADA0F7961
http://purl.uniprot.org/uniprot/Q05CT3http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/2B9E0E0730A10F9D7C199D3C6F674C72E5581DE09D3A995B52C34A56D4CEA55B14683D5072B5FB3F4CA15CCADA0F7961
http://purl.uniprot.org/uniprot/#_Q05CT3-mappedCitation-32011034http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/2B9E0E0730A10F9D7C199D3C6F674C72E5581DE09D3A995B52C34A56D4CEA55B14683D5072B5FB3F4CA15CCADA0F7961