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DESCRIBE <http://purl.uniprot.org/SHA-384/2D0D5EE75C5454AFACB651EB4684A3E9D0CA6E3C91240295E1815A3A4C8831F601CDB9259EBC3B762E51182B25E814DD>
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http://purl.uniprot.org/SHA-384/2D0D5EE75C5454AFACB651EB4684A3E9D0CA6E3C91240295E1815A3A4C8831F601CDB9259EBC3B762E51182B25E814DD
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/2D0D5EE75C5454AFACB651EB4684A3E9D0CA6E3C91240295E1815A3A4C8831F601CDB9259EBC3B762E51182B25E814DD
http://www.w3.org/2000/01/rdf-schema#comment
"miR-130a plays a critical role in attenuation of cardiac dysfunction and remodeling after myocardial infarction. The mechanisms involve activation of PI3K/Akt signaling via suppression of PTEN expression."
xsd:string
http://purl.uniprot.org/uniprot/#_C7C88F32DD47511F113F6A53DA5EF4FBD0C5C2BFEF87AF7EF033B38EF7CC4A384A20DF8B4CC84AD953745162226E6D9B
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/2D0D5EE75C5454AFACB651EB4684A3E9D0CA6E3C91240295E1815A3A4C8831F601CDB9259EBC3B762E51182B25E814DD
http://purl.uniprot.org/uniprot/F6KCZ4
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/2D0D5EE75C5454AFACB651EB4684A3E9D0CA6E3C91240295E1815A3A4C8831F601CDB9259EBC3B762E51182B25E814DD
http://purl.uniprot.org/uniprot/#_F6KCZ4-mappedCitation-26458524
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/2D0D5EE75C5454AFACB651EB4684A3E9D0CA6E3C91240295E1815A3A4C8831F601CDB9259EBC3B762E51182B25E814DD