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DESCRIBE <http://purl.uniprot.org/SHA-384/2DB68D0E5655A08925B9DD75F217D8B73D90044E057567E7C39FB05F3D4FCFC5B5D2F38BA43A777F80934623267C76AC>
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http://purl.uniprot.org/SHA-384/2DB68D0E5655A08925B9DD75F217D8B73D90044E057567E7C39FB05F3D4FCFC5B5D2F38BA43A777F80934623267C76AC
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/2DB68D0E5655A08925B9DD75F217D8B73D90044E057567E7C39FB05F3D4FCFC5B5D2F38BA43A777F80934623267C76AC
http://www.w3.org/2000/01/rdf-schema#comment
"The activation of p38 in response to low doses of ultraviolet radiation was postulated to be protective for p53-inactive cells. Therefore MCPIP1 may favor the survival of p53-defective HaCaT cells by sustaining the activation of p38."
xsd:string
http://purl.uniprot.org/uniprot/#_31D8EEC95ED1CA3EA3B491B6CEF4A29019DDEB194E49539320F4B57EEFEDA2DFFE04230DE7A52E53F7472B8901601759
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/2DB68D0E5655A08925B9DD75F217D8B73D90044E057567E7C39FB05F3D4FCFC5B5D2F38BA43A777F80934623267C76AC
http://purl.uniprot.org/uniprot/Q5D1E8
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/2DB68D0E5655A08925B9DD75F217D8B73D90044E057567E7C39FB05F3D4FCFC5B5D2F38BA43A777F80934623267C76AC
http://purl.uniprot.org/uniprot/#_Q5D1E8-mappedCitation-29103983
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/2DB68D0E5655A08925B9DD75F217D8B73D90044E057567E7C39FB05F3D4FCFC5B5D2F38BA43A777F80934623267C76AC