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DESCRIBE <http://purl.uniprot.org/SHA-384/2EB0ADEF32B327EE87471C5386D8F907931768FABAFCC1ABB5885B0C680C8CA76E443F03D5007DEA5C598362D09FBB81>
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http://purl.uniprot.org/SHA-384/2EB0ADEF32B327EE87471C5386D8F907931768FABAFCC1ABB5885B0C680C8CA76E443F03D5007DEA5C598362D09FBB81
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/2EB0ADEF32B327EE87471C5386D8F907931768FABAFCC1ABB5885B0C680C8CA76E443F03D5007DEA5C598362D09FBB81
http://www.w3.org/2000/01/rdf-schema#comment
"Defective p53 signaling in p53 wild-type tumors attenuates p21waf1 induction and cyclin B repression rendering them sensitive to Chk1 inhibitors that abrogate DNA damage-induced S and G2 arrest."
xsd:string
http://purl.uniprot.org/uniprot/#_9841FFB2A189A8608EC8471FE98836FDB105E09E4CD0EC7F4A88C031CA1E0767395BB17F35077AA72C861C393948FEBF
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/2EB0ADEF32B327EE87471C5386D8F907931768FABAFCC1ABB5885B0C680C8CA76E443F03D5007DEA5C598362D09FBB81
http://purl.uniprot.org/uniprot/A0A346XLP3
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/2EB0ADEF32B327EE87471C5386D8F907931768FABAFCC1ABB5885B0C680C8CA76E443F03D5007DEA5C598362D09FBB81
http://purl.uniprot.org/uniprot/#_A0A346XLP3-mappedCitation-18281511
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/2EB0ADEF32B327EE87471C5386D8F907931768FABAFCC1ABB5885B0C680C8CA76E443F03D5007DEA5C598362D09FBB81