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DESCRIBE <http://purl.uniprot.org/SHA-384/3267F4C8649ADA044E85ABC3D22AE899807696C8D8A3FD20F86E72AB5B4385CE390E30E0AF0385D93BDC810CD7FD2269>
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http://purl.uniprot.org/SHA-384/3267F4C8649ADA044E85ABC3D22AE899807696C8D8A3FD20F86E72AB5B4385CE390E30E0AF0385D93BDC810CD7FD2269
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/3267F4C8649ADA044E85ABC3D22AE899807696C8D8A3FD20F86E72AB5B4385CE390E30E0AF0385D93BDC810CD7FD2269
http://www.w3.org/2000/01/rdf-schema#comment
"Novel Dst(Gt) mice showed that a loss-of-function mutation in the actin-binding domain-containing Dystonin isoforms led to typical dystonia musculorum phenotypes"
xsd:string
http://purl.uniprot.org/uniprot/#_0CAF08FB6E1FBDD964C31E9579449D8C00B5A399CD9618D74AE9F1A41DB91A61DEF67A810F21BD4202E5C5A7333CF64F
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/3267F4C8649ADA044E85ABC3D22AE899807696C8D8A3FD20F86E72AB5B4385CE390E30E0AF0385D93BDC810CD7FD2269
http://purl.uniprot.org/uniprot/Q91ZU6
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/3267F4C8649ADA044E85ABC3D22AE899807696C8D8A3FD20F86E72AB5B4385CE390E30E0AF0385D93BDC810CD7FD2269
http://purl.uniprot.org/uniprot/#_Q91ZU6-mappedCitation-25195653
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/3267F4C8649ADA044E85ABC3D22AE899807696C8D8A3FD20F86E72AB5B4385CE390E30E0AF0385D93BDC810CD7FD2269