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DESCRIBE <http://purl.uniprot.org/SHA-384/335541F241A19A52456F1382B5BCBA1D0AAA31193A8138CBE5E03F8CD504A67BC067830BFA4C83811A64EE9B781DEFBA>
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http://purl.uniprot.org/SHA-384/335541F241A19A52456F1382B5BCBA1D0AAA31193A8138CBE5E03F8CD504A67BC067830BFA4C83811A64EE9B781DEFBA
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/335541F241A19A52456F1382B5BCBA1D0AAA31193A8138CBE5E03F8CD504A67BC067830BFA4C83811A64EE9B781DEFBA
http://www.w3.org/2000/01/rdf-schema#comment
"Results suggest that activation of JNK in Alzheimer's disease (AD) inhibits insulin signaling which could lead to a decreased expression of VGLUT1 therefore contributing to the glutamatergic deficit in AD"
xsd:string
http://purl.uniprot.org/uniprot/#_698F2C33460E662464D089CA7FA214972E292BB22778F2AB689077E827A8993EDEFD02FDAB89C02BE7FA5E97266305C3
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/335541F241A19A52456F1382B5BCBA1D0AAA31193A8138CBE5E03F8CD504A67BC067830BFA4C83811A64EE9B781DEFBA
http://purl.uniprot.org/uniprot/Q9P2U7
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/335541F241A19A52456F1382B5BCBA1D0AAA31193A8138CBE5E03F8CD504A67BC067830BFA4C83811A64EE9B781DEFBA
http://purl.uniprot.org/uniprot/#_Q9P2U7-mappedCitation-26836159
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/335541F241A19A52456F1382B5BCBA1D0AAA31193A8138CBE5E03F8CD504A67BC067830BFA4C83811A64EE9B781DEFBA